Jerry replies to J B who accuses Jerry of "making this up" re hypermobility syndrome.

 DECEMBER 26, 2012 - 10:25
AM  jerry   hesch
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 Jerry Hesch MHS, PT from the
  Hesch Institute replies to JB
JB: OCTOBER 4,
2012 –9:16 PM  JB

JB writes: 
 Jerry At 6:10 you state, “The   asian population tends more to be hypermobile. That’s well established in the  literature.” You completely made this up. I am familiar with one study in the  70′s that compared American vs. Indian hip ROM and found that Indians had more  hip IR, but know of no other studies comparing cultural mobility. This video  further supports the views expressed on this
site.
 
Jerry Hesch replies: 
It is well established that there are populations such as Asians in which
  hypermobility. Here are some  references.
 Why do make deprecatory  remarks such as “You completely made this
up.”

 “BHJS seems to occur more
often in Asian-American children than in Caucasian children, and it is least
common in African-American children.”
http://www.webmd.com/rheumatoid-arthritis/benign-hypermobility-joint-syndrome

Remvig L. Jensen DV. Ward
RC., Epidemiology of general joint hypermobility and basis for the proposed
criteria for benign joint hypermobility syndrome: review of the literature. J
Rheumatol April 2007 34(4):804-809
(1A)

PHYS THER. 1999;
79:591-599. Leslie N
Russek.  Hypermobility
Syndrome 


Grahame R. “The
hypermobility syndrome.” Ann Rheum Dis.
1990; 49:190
–200.

Wordsworth P, Ogilvie D,
Smith R, Sykes B.Joint mobility with particular reference to racial variation
and inherited connective tissue disorders. Br J Rheumatol. 1987
Feb;26(1):9-12.

 Abstract

Joint mobility scores were
  compared in 248 normal English Caucasian males and females between the ages of
  8 and 70 years. The results were contrasted with those in a group of normal
  Asian Indians and patients suffering from a variety of inherited disorders
  including Type II Ehlers-Danlos syndrome (EDS), Type I osteogenesis imperfecta
  (OI), Marfan syndrome, generalized osteoarthritis (GOA), achondroplasia and
  pseudoachondroplasia. The first-degree relatives of ten subjects with severe or
  lethal OI were also examined. Asian Indians were significantly more mobile than
  English Caucasians. Males and females with EDS were hypermobile but only the
  females with OI and the female relatives of those with severe or lethal OI
  showed excess joint laxity. Patients with pseudoachondroplasia exhibited a
  grosser type of joint laxity than other subjects while those with GOA
  represented a relatively stiff group. No evidence was found to support the view
  that hypermobility is associated with particular length variants at the Type II
  collagen structural gene locus
(COL2A1).

 Everman DB, Robin NH.
  Hypermobility syndrome. Pediatr Rev. 1998; 19:111
–117.  

Finsterbush A, Pogrund H.
The hypermobility syndrome. Clin Orthop Relat Res. May1982 :
124–127.

Larsson LG, Baum J,
Mudholkar GS, Kollia GD. Benefits and disadvantages of joint hypermobility among
musicians. N Engl J Med. 1993;329:1079–1082. Available at:
  http://content.nejm.org/cgi/content/full/329/15/1079. Accessed August 29,
2006. 

SimpsonMR, Benign Joint
  Hypermobility Syndrome: Evaluation, Diagnosis, and
Management

 J Am Osteopath Assoc
September 1, 2006 vol. 106 no. 9
531-536  

Louise J Tofts,1,2,3,4
Elizabeth J Elliott,4,5,6 Craig Munns,1,4,7 Verity Pacey,1,3,4,8 and David O
Sillence1 The differential diagnosis of children with joint hypermobility: a
review of the literature Pediatr Rheumatol Online J. 2009; 7: 1. Published
online 2009 January 5. doi: 10.1186/1546-0096-7-1