DECEMBER 26, 2012 - 10:25
AM
jerry
hesch
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Jerry Hesch MHS, PT from the
Hesch Institute replies to Joseph
Brence
Joseph Brence: OCTOBER 4,
2012 –9:16 PM
josephbrence
At 6:10 you state, “The
asian population tends more to be hypermobile. That’s well established in the
literature.” You completely made this up. I am familiar with one study in the
70′s that compared American vs. Indian hip ROM and found that Indians had more
hip IR, but know of no other studies comparing cultural mobility. This video
further supports the views expressed on this
site.
Jerry Hesch: Joseph Brence,
It is well established that there are populations such as Asians in which
hypermobility. Here are some
references.
Why do make deprecatory
remarks such as “You completely made this
up.” Is that a result of you
education or your upbringing, personality, or
other?
Having seen your posts on
other sites, this is merely a rhetorical question. It is so unnecessary, but it
is your choice.
Here is a direct quote form the contemporary literature, with several references listed.
“BHJS seems to occur more
often in Asian-American children than in Caucasian children, and it is least
common in African-American children.”
http://www.webmd.com/rheumatoid-arthritis/benign-hypermobility-joint-syndrome
Remvig L. Jensen DV. Ward
RC., Epidemiology of general joint hypermobility and basis for the proposed
criteria for benign joint hypermobility syndrome: review of the literature. J
Rheumatol April 2007 34(4):804-809
(1A)
PHYS THER. 1999;
79:591-599. Leslie N
Russek. Hypermobility
Syndrome.
Grahame R. “The
hypermobility syndrome.” Ann Rheum Dis.
1990; 49:190
–200.
Wordsworth P, Ogilvie D,
Smith R, Sykes B.Joint mobility with particular reference to racial variation
and inherited connective tissue disorders. Br J Rheumatol. 1987
Feb;26(1):9-12.
Abstract
Joint mobility scores were
compared in 248 normal English Caucasian males and females between the ages of
8 and 70 years. The results were contrasted with those in a group of normal
Asian Indians and patients suffering from a variety of inherited disorders
including Type II Ehlers-Danlos syndrome (EDS), Type I osteogenesis imperfecta
(OI), Marfan syndrome, generalized osteoarthritis (GOA), achondroplasia and
pseudoachondroplasia. The first-degree relatives of ten subjects with severe or
lethal OI were also examined. Asian Indians were significantly more mobile than
English Caucasians. Males and females with EDS were hypermobile but only the
females with OI and the female relatives of those with severe or lethal OI
showed excess joint laxity. Patients with pseudoachondroplasia exhibited a
grosser type of joint laxity than other subjects while those with GOA
represented a relatively stiff group. No evidence was found to support the view
that hypermobility is associated with particular length variants at the Type II
collagen structural gene locus
(COL2A1).
Everman DB, Robin NH.
Hypermobility syndrome. Pediatr Rev. 1998; 19:111
–117.
Finsterbush A, Pogrund H.
The hypermobility syndrome. Clin Orthop Relat Res. May1982 :
124–127.
Larsson LG, Baum J,
Mudholkar GS, Kollia GD. Benefits and disadvantages of joint hypermobility among
musicians. N Engl J Med. 1993;329:1079–1082. Available at:
http://content.nejm.org/cgi/content/full/329/15/1079. Accessed August 29,
2006.
SimpsonMR, Benign Joint
Hypermobility Syndrome: Evaluation, Diagnosis, and
Management. J Am Osteopath Assoc
September 1, 2006 vol. 106 no. 9
531-536
Louise J Tofts,1,2,3,4
Elizabeth J Elliott,4,5,6 Craig Munns,1,4,7 Verity Pacey,1,3,4,8 and David O
Sillence1 The differential diagnosis of children with joint hypermobility: a
review of the literature Pediatr Rheumatol Online J. 2009; 7: 1. Published
online 2009 January 5. doi: 10.1186/1546-0096-7-1